Studies on the cytotoxic mechanisms of ginkgetin in a human ovarian adenocarcinoma cell line

Keyphrases

• Adenocarcinoma Cells 100%
• Cytotoxic Mechanism 100%
• Ovarian Adenocarcinoma 100%
• Ginkgetin 100%
• Catalase 37%
• Biflavone 37%
• Hydrogen Peroxide 25%
• DNA Fragmentation 25%
• HeLa 25%
• Caspase 25%
• Protective Effect 25%
• OVCAR-3 25%
• Z-VAD-FMK 25%
• Autoxidation 12%
• Apoptosis 12%
• Induced Apoptosis 12%
• Cytotoxicity 12%
• Caspase-3 12%
• Cell Death 12%
• Pre-treating 12%
• Incubation 12%
• Drug Treatment 12%
• Morphological Changes 12%
• Antioxidant 12%
• Caspase Inhibitor 12%
• Cytotoxic Effect 12%
• MTT Assay 12%
• Human Cell Lines 12%
• Vitamin C 12%
• Intracellular Level 12%
• Skin Fibroblasts 12%
• EC50 12%
• 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium Bromide 12%
• Vitamin E 12%
• Cervical Carcinoma 12%
• DNA Breaks 12%
• DNA Breakage 12%
• Selaginella Moellendorffii 12%

Pharmacology, Toxicology and Pharmaceutical Science

• Catalase 100%
• Ovary Adenocarcinoma 100%
• Hydrogen Peroxide 66%
• Caspase 66%
• Cytotoxicity 66%
• Caspase 3 33%
• Antioxidant 33%
• Ascorbic Acid 33%
• Pharmacotherapy 33%
• 3 (4,5 Dimethyl 2 Thiazolyl) 2,5 Diphenyltetrazolium Bromide 33%
• Caspase Inhibitor 33%
• Alpha Tocopherol 33%
• Uterine Cervix Carcinoma 33%
• Selaginella moellendorffii 33%
• EC50 33%