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Pathogenesis of α-Synuclein in Parkinson’s Disease: From a Neuron-Glia Crosstalk Perspective
Shuanglong Yi
, Linfang Wang
, Honglei Wang
,
Margaret S. Ho
*
, Shiping Zhang
*
*
此作品的通信作者
神經科學研究所
研究成果
:
Review article
›
同行評審
84
引文 斯高帕斯(Scopus)
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Keyphrases
Parkinson's Disease
100%
Alpha-synuclein (α-syn)
100%
Glia
57%
Glial Activation
57%
Dopaminergic Neurons
42%
Clinical Treatment
14%
Cognitive Impairment
14%
Simply Supported
14%
Neuroinflammation
14%
Disease Progression
14%
Tremor
14%
Neurodegeneration
14%
Bystander
14%
Neuronal Function
14%
Neurodegenerative Diseases
14%
Dysregulation
14%
Parkinson Patients
14%
Neuronal Development
14%
Disease Onset
14%
Progressive Neurodegeneration
14%
Substantia Nigra
14%
Parkinson's Disease Treatment
14%
Lewy Bodies
14%
Lewy Neurites
14%
Non-motor Symptoms
14%
Iron Deposition
14%
Motor Symptoms
14%
Bradykinesia
14%
Disease Pathology
14%
Preclinical Therapy
14%
Ferroptosis
14%
Anosmia
14%
Brain Physiology
14%
Neuroscience
Parkinson's Disease
100%
Glia
100%
Synuclein
100%
Dopaminergic Neuron
37%
Neurodegenerative Disorder
25%
Neurite
12%
Neural Development
12%
Cognitive Disorders
12%
Tremor
12%
Nerve Cell Degeneration
12%
Substantia nigra
12%
Lewy Body
12%
Non-Motor Symptoms
12%
Bradykinesia
12%
Anosmia
12%
Pathology of Parkinson's Disease
12%
Ferroptosis
12%