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Murinization of Internalin Extends Its Receptor Repertoire, Altering Listeria monocytogenes Cell Tropism and Host Responses
Yu Huan Tsai
, Olivier Disson, Hélène Bierne, Marc Lecuit
*
*
此作品的通信作者
微生物及免疫學研究所
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引文 斯高帕斯(Scopus)
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Keyphrases
Listeria Monocytogenes
100%
Host Response
100%
Cell Tropism
100%
Internalin
100%
E-cadherin
63%
InlA
45%
Humanized Mice
27%
N-cadherin
18%
Wild Mice
18%
Pathogenic Properties
18%
Knock-in Mouse Model
9%
Innate Immune Response
9%
Foodborne Pathogens
9%
Transgenic Mice
9%
Central Nervous System
9%
Surface Proteins
9%
Human Pathogens
9%
M Cells
9%
Intestinal Barrier
9%
Listeriosis
9%
Host Range
9%
Goblet Cells
9%
Intestinal Epithelium
9%
Host Receptor
9%
Portal of Entry
9%
Intestinal Barrier Injury
9%
Maternal-fetal Infection
9%
Human Listeriosis
9%
Immunology and Microbiology
Immune Response
100%
Listeria monocytogenes
100%
Internalin
100%
Cadherin
63%
Humanized Mouse
27%
Neural Cell Adhesion Molecule
18%
Wild Type Mouse
18%
Internalization
18%
Listeriosis
18%
Wild Type
9%
Infectious Agent
9%
Innate Immune System
9%
Host Range
9%
Central Nervous System
9%
Transgenic Mouse
9%
Human Pathogen
9%
Goblet Cell
9%