H-Ras exerts opposing effects on type I interferon responses depending on its activation status

Keyphrases

• HEC-RAS 100%
• Activation Status 100%
• Opposing Effects 100%
• Type I Interferon Response 100%
• RLR Signaling 41%
• Sendai Virus 25%
• Further Analysis 8%
• Negative Effects 8%
• Promoter Activity 8%
• Inhibitory Effect 8%
• Antiviral Activity 8%
• Ectopic Expression 8%
• High-throughput Screening 8%
• Signaling Activity 8%
• Mitochondria 8%
• Virus Replication 8%
• Short Hairpin RNA (shRNA) 8%
• Signaling Complex 8%
• High-level Expression 8%
• Virus Infection 8%
• Retinoic Acid-inducible Gene I (RIG-I) 8%
• Ras Proteins 8%
• Signaling Regulation 8%
• Sindbis Virus 8%
• Type I Interferon Signaling 8%
• Vesicular Stomatitis Virus 8%
• Signalosome 8%
• RasV12 8%
• Type I IFN 8%
• TNF Receptor-associated Factor 3 8%

Biochemistry, Genetics and Molecular Biology

• Sendai Virus 100%
• Interferon Type I 100%
• Wild Type 33%
• Promoter Region 33%
• Virus Replication 33%
• Ectopic Expression 33%
• Small Hairpin RNA 33%
• Mitochondrion 33%
• Tretinoin 33%
• Retinoic Acid 33%
• High Throughput Screening 33%
• TNF Receptor Associated Factor 33%
• Antiviral Activity 33%
• Vesiculovirus 33%
• Sindbis Virus 33%