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Galectin-1 upregulates CXCR4 to promote tumor progression and poor outcome in kidney cancer
Chang Shuo Huang
, Shye Jye Tang
, Ling Yen Chung
, Cheng Ping Yu
, Jar Yi Ho
, Tai Lung Cha
, Chii Cheng Hsieh
, Hsiao Hsien Wang
, Guang Huan Sun
*
,
Kuang Hui Sun
*
此作品的通信作者
醫學生物技術暨檢驗學系
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引文 斯高帕斯(Scopus)
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Keyphrases
Tumor Progression
100%
Galectin-1 (Gal-1)
100%
Poor Outcome
100%
C-X-C Chemokine Receptor Type 4 (CXCR4)
100%
Kidney Cancer
100%
CXCR4 Expression
28%
Renal Cell Carcinoma
21%
Angiogenesis
14%
Knockdown
14%
Expression Level
14%
Clonogenic
14%
Kidney Cancer Cell
14%
Clinical Significance
7%
Tumor
7%
Epithelial-mesenchymal Transition
7%
Gene Expression
7%
Prognostic Factors
7%
Cell Invasion
7%
Clinical Prognosis
7%
Nuclear factor-B
7%
Cell Differentiation
7%
Lectin
7%
Cell Adhesion
7%
NF-B
7%
Therapeutic Target
7%
Cancer Cell Lines
7%
Galactosides
7%
Disease-free Survival
7%
Physiological Processes
7%
Reduced Cell
7%
Cell Tumor
7%
Cell Motility
7%
Tissue Specimens
7%
Pathologic Processes
7%
Renal Cancer Cells
7%
Biochemistry, Genetics and Molecular Biology
Tumor Progression
100%
Galectin-1
100%
CXCR4
100%
Cancer Cell
21%
Upregulation
14%
Angiogenesis
14%
Gene Expression
7%
Mesenchymal-Epithelial Transition
7%
Cell Adhesion
7%
Cell Migration
7%
Cell Invasion
7%
Disease Free Survival
7%
Complement Factor B
7%
Cell Motility
7%
Lectin Binding
7%
Galactoside
7%