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Effects of signalling transduction modulators on the transformed phenotypes in v-H-ras-transformed NIH 3T3 cells
Min Liang Kuo
*
,
Jaw Jou Kang
, Nae Cherng Yang
*
此作品的通信作者
食品安全及健康風險評估研究所
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3
引文 斯高帕斯(Scopus)
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深入研究「Effects of signalling transduction modulators on the transformed phenotypes in v-H-ras-transformed NIH 3T3 cells」主題。共同形成了獨特的指紋。
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Keyphrases
NIH3T3
100%
HEC-RAS
100%
Signal Transduction
100%
Transformed Phenotype
100%
Protein Kinase
75%
Cellular Growth
75%
Transformant
50%
Soft Agar
50%
Phospholipase A2 (PLA2)
50%
Cyclooxygenase-1 (COX-1)
50%
Phospholipase C
50%
Focus Formation
50%
Signaling Pathway
25%
Tyrosine Kinase Inhibitor
25%
Aristolochic Acid
25%
Genistein
25%
Tyrosine Kinase
25%
Morphological Development
25%
Micromolar Concentration
25%
Cyclic Adenosine Monophosphate (cAMP)
25%
Dose-ranging
25%
Anchorage-independent Growth
25%
Cellular Morphology
25%
Transformation Events
25%
Forskolin
25%
PKC Inhibitor
25%
3-Isobutyl-1-methylxanthine
25%
Kanamycin Sulfate
25%
Compound Inhibitor
25%
Signaling Modulators
25%
Tyrphostin
25%
Pharmacology, Toxicology and Pharmaceutical Science
Protein Tyrosine Kinase
100%
Protein Kinase C
100%
Agar
100%
Prostaglandin Synthase
100%
Phospholipase A2
100%
Lecithinase
100%
Protein Tyrosine Kinase Inhibitor
50%
Aristolochic Acid
50%
Genistein
50%
Cyclic AMP Dependent Protein Kinase
50%
Cyclic AMP
50%
Forskolin
50%
Isobutylmethylxanthine
50%
Tyrphostin
50%
Neomycin
50%
Neuroscience
Cell Growth
100%
Agar
66%
Protein Kinase C
66%
Cyclooxygenase
66%
Phospholipase A2
66%
Lecithinase
66%
Protein Kinase A
33%
Cyclic Adenosine Monophosphate
33%
Receptor Tyrosine Kinase
33%
Tyrosine Kinase
33%
Forskolin
33%
Aristolochic Acid
33%
Tyrosine Kinase Inhibitor
33%
IBMX
33%
Anchorage Independent Growth
33%
Genistein
33%