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Aurora-A overexpression enhances cell-aggregation of Ha-ras transformants through the MEK/ERK signaling pathway.
Ya Shih Tseng
*
, Jenq Chang Lee,
Chi Ying F. Huang
, Hsiao Sheng Liu
*
此作品的通信作者
生物藥學研究所
研究成果
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21
引文 斯高帕斯(Scopus)
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Keyphrases
Overexpression
100%
Transformant
100%
ERK Pathway
100%
Aurora A
100%
Cell Aggregation
100%
Ha-ras
100%
RasV12
63%
Focus Formation
36%
Human Bladder Cancer
18%
Dominant Negative
9%
Phosphorylation
9%
Cancer Tissue
9%
Signaling Pathway
9%
Immunohistochemical Staining
9%
Induced Aggregation
9%
Small Interfering RNA (siRNA)
9%
Colon Cancer
9%
Overexpressing
9%
Akt Phosphorylation
9%
3T3 Fibroblasts
9%
Tumorigenesis
9%
Sequence Analysis
9%
Cellular Response
9%
Single-molecule Sequencing
9%
Proliferation Rate
9%
Pharmacological Inhibitors
9%
KRAS mutation
9%
Bladder Cancer
9%
Real-time PCR Analysis
9%
Ki-ras
9%
Colon Tissue
9%
RAS Pathway
9%
MEK-ERK
9%
Human Colon Cancer
9%
Acting Together
9%
Medicine and Dentistry
Ras Signaling
100%
Cell Signaling Pathway
100%
Cell Aggregation
100%
Bladder Cancer
50%
Cancer Tissue
33%
Colon Carcinoma
33%
Cell Line
16%
Protein Sequencing
16%
Sequence Analysis
16%
Immunohistochemistry
16%
Carcinogenesis
16%
Small Interfering RNA
16%
Phosphotransferase
16%
Real Time Polymerase Chain Reaction
16%
Fibroblast
16%
Biochemistry, Genetics and Molecular Biology
Wild Type
100%
Cell Aggregation
100%
Colon
50%
Real-Time Polymerase Chain Reaction
25%
Protein Sequencing
25%
Phosphotransferase
25%
Kinase
25%
Carcinogenesis
25%
Small Interfering RNA
25%
Shotgun Sequencing
25%
Fibroblast
25%
Pharmacology, Toxicology and Pharmaceutical Science
Bladder Cancer
100%
Colon Carcinoma
66%
Carcinogenesis
33%
Phosphotransferase
33%
Small Interfering RNA
33%