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Aquatic birnavirus capsid protein, VP3, induces apoptosis via the Bad-mediated mitochondria pathway in fish and mouse cells
Chien Li Chiu, Jen Leih Wu,
Guor Mour Her
, Yi Li Chou, Jiann Ruey Hong
*
*
此作品的通信作者
生物藥學研究所
研究成果
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同行評審
28
引文 斯高帕斯(Scopus)
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Keyphrases
Induced Apoptosis
100%
Mitochondrial Pathway
100%
Capsid Protein
100%
Mouse Cells
100%
Aquatic Birnaviruses
100%
Fish Cell Line
100%
Caspase-3
50%
Overexpression
50%
Apoptotic Cell Death
50%
Mitochondrial Membrane Potential Loss
50%
CHSE-214
50%
Mitochondrial Function
25%
Caspase-3 Activation
25%
Gene Expression
25%
Cell Viability
25%
NIH3T3
25%
Encoded Protein
25%
Zebrafish
25%
Western Blot Analysis
25%
Viral Genome
25%
Caspase-9
25%
Anti-apoptotic Proteins
25%
Viral Replication
25%
Apoptotic Genes
25%
Mitochondrial Damage
25%
TUNEL Assay
25%
Antisense RNA
25%
Post-infection
25%
Necrotic Cell Death
25%
Death Pathway
25%
Effector Caspases
25%
Death Process
25%
Initiator Caspase
25%
Mitochondrial Disruption
25%
VP3 Gene
25%
Immunology and Microbiology
Caspase
100%
Capsid
100%
Mitochondrion
100%
Birnaviridae
100%
Programmed Cell Death
100%
Upregulation
50%
Genetic Transfection
50%
Mitochondrial Membrane Potential
50%
Viral Replication
25%
Cell Death
25%
Gene Expression
25%
Western Blot
25%
Cell Viability
25%
Zebra Fish
25%
Antisense
25%
Virus Genome
25%
Neuroscience
Mitochondrion
100%
Protein VP3
100%
Birnaviridae
100%
Programmed Cell Death
100%
Caspase 3
75%
Transfection
50%
Mitochondrial Membrane Potential
50%
Gene Expression
25%
Western Blot
25%
Cell Viability
25%
Cell Death
25%
TUNEL Assay
25%
Caspase-9
25%
Antisense RNA
25%
Initiator Caspase
25%
Virus Genome
25%
Effector Caspase
25%