Temporal expression patterns of distinct cytokines and M1/M2 macrophage polarization regulate rheumatoid arthritis progression

Keyphrases

• Rheumatoid Arthritis 100%
• M2 Polarization 100%
• M2 Macrophages 100%
• M2 Macrophage Polarization 100%
• Temporal Expression Pattern 100%
• Chronic Inflammation 50%
• Inflammatory Cytokines 50%
• Switching Time 50%
• Interleukin-17 (IL-17) 50%
• Chronic Pain 50%
• Tumor Necrosis Factor-α 50%
• Anti-inflammatory Cytokines 50%
• T-cell Death-associated Gene 8 50%
• Inflammation 25%
• Autoimmune Disease 25%
• Mouse Model 25%
• Polarization Switching 25%
• Interleukin-6 25%
• Acute Stage 25%
• Dione 25%
• Temporal Expression 25%
• Immune Infiltration 25%
• Cytokine Production 25%
• 1,2,5-thiadiazole 25%
• Maintenance Stage 25%
• M1 Macrophages 25%
• Joint Destruction 25%
• Joint Inflammation 25%
• Established Rheumatoid Arthritis 25%
• Synoviocytes 25%
• Synovial Joint 25%
• Interleukin-6 Expression 25%
• Chronic Joint Inflammation 25%
• Resident Macrophages 25%

Immunology and Microbiology

• Inflammatory Arthritis 100%
• Cytokine 100%
• M2 Macrophages 100%
• Macrophage 40%
• Proinflammatory Cytokine 40%
• T Cell 40%
• Tumor Necrosis Factor 40%
• Cell Death 40%
• Inflammatory Cytokine 40%
• Interleukin 6 40%
• Arthritis 40%
• Interleukin 17 40%
• Autoimmune Disease 20%
• Mouse Model 20%
• Immunocompetent Cell 20%
• Cytokine Production 20%
• Synoviocyte 20%