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PML, a growth suppressor disrupted in acute promyelocytic leukemia
Zhao Mei Mu
, Khew Voon Chin
, Jin Hwang Liu
, Guillermina Lozano
, Kun Sang Chang
*
*
Corresponding author for this work
Institute of Biopharmaceutical Sciences
Research output
:
Contribution to journal
›
Article
›
peer-review
300
Scopus citations
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Keyphrases
Acute Promyelocytic Leukemia
100%
Retinoic Acid Receptor
100%
Fusion Protein
28%
Dominant Negative
28%
Co-transfection
28%
NIH3T3
14%
Oncogene
14%
Fibroblasts
14%
Tumorigenicity
14%
Nude Mice
14%
Subcellular Localization
14%
Soft Agar
14%
Promoter-specific
14%
Anchorage-independent Growth
14%
Chromosomal Translocation
14%
Zinc Finger Transcription Factor
14%
NB4 Cells
14%
Acute Promyelocytic Leukemia Cells
14%
Leukemogenesis
14%
Oncogenic Transformation
14%
Rat Embryo
14%
Biochemistry, Genetics and Molecular Biology
Polymorphonuclear Cell
100%
Retinoic Acid Receptor
35%
Promyelocytic Leukemia Protein
10%
Chimeric Protein
10%
Promoter Region
5%
Transcription
5%
Carcinogenesis
5%
Oncogene
5%
Subcellular Localization
5%
Nude Mouse
5%
Agar
5%
Anchorage Independent Growth
5%
Chromosomal Translocation
5%
Zinc Finger Transcription Factor
5%
Oncogene Neu
5%
Fibroblast
5%
Rat
5%