HOming in on arteriovenous fistula survival

W. Durante; C. C. Lin

doi:10.1038/ki.2008.127

HOming in on arteriovenous fistula survival

W. Durante^*, C. C. Lin

^*Corresponding author for this work

Department of Internal Medicine

Research output: Contribution to journal › Comment/debate

13 Scopus citations

Abstract

Vascular access failure is a major clinical problem for patients on hemodialysis, but therapeutic approaches aimed at improving arteriovenous fistula (AVF) dysfunction remain elusive. Using a murine AVF model, Juncos et al. demonstrate for the first time that the stress protein heme oxygenase-1 (HO-1) is induced in AVFs and that HO-1 is a critical determinant of AVF survival.

Original language	English
Pages (from-to)	9-11
Number of pages	3
Journal	Kidney International
Volume	74
Issue number	1
DOIs	https://doi.org/10.1038/ki.2008.127
State	Published - Jul 2008

Access to Document

10.1038/ki.2008.127

Cite this

@article{e0cbe7b225a548ad92442f62179b81b8,

title = "HOming in on arteriovenous fistula survival",

abstract = "Vascular access failure is a major clinical problem for patients on hemodialysis, but therapeutic approaches aimed at improving arteriovenous fistula (AVF) dysfunction remain elusive. Using a murine AVF model, Juncos et al. demonstrate for the first time that the stress protein heme oxygenase-1 (HO-1) is induced in AVFs and that HO-1 is a critical determinant of AVF survival.",

author = "W. Durante and Lin, {C. C.}",

note = "Funding Information: The authors are supported, in part, by grants from the National Institutes of Health (HL59976, HL749976), the Taipei Veterans General Hospital (V95-ER-003, V96-ER-B2-009), and the National Science Council of Taiwan (NSC95-2314-B-075-070, NSC96-2314-B-010-045).",

year = "2008",

month = jul,

doi = "10.1038/ki.2008.127",

language = "English",

volume = "74",

pages = "9--11",

journal = "Kidney International",

issn = "0085-2538",

publisher = "Elsevier Inc.",

number = "1",

}

TY - JOUR

T1 - HOming in on arteriovenous fistula survival

AU - Durante, W.

AU - Lin, C. C.

N1 - Funding Information: The authors are supported, in part, by grants from the National Institutes of Health (HL59976, HL749976), the Taipei Veterans General Hospital (V95-ER-003, V96-ER-B2-009), and the National Science Council of Taiwan (NSC95-2314-B-075-070, NSC96-2314-B-010-045).

PY - 2008/7

Y1 - 2008/7

N2 - Vascular access failure is a major clinical problem for patients on hemodialysis, but therapeutic approaches aimed at improving arteriovenous fistula (AVF) dysfunction remain elusive. Using a murine AVF model, Juncos et al. demonstrate for the first time that the stress protein heme oxygenase-1 (HO-1) is induced in AVFs and that HO-1 is a critical determinant of AVF survival.

AB - Vascular access failure is a major clinical problem for patients on hemodialysis, but therapeutic approaches aimed at improving arteriovenous fistula (AVF) dysfunction remain elusive. Using a murine AVF model, Juncos et al. demonstrate for the first time that the stress protein heme oxygenase-1 (HO-1) is induced in AVFs and that HO-1 is a critical determinant of AVF survival.

UR - http://www.scopus.com/inward/record.url?scp=45249090575&partnerID=8YFLogxK

U2 - 10.1038/ki.2008.127

DO - 10.1038/ki.2008.127

M3 - Comment/debate

C2 - 18560362

AN - SCOPUS:45249090575

SN - 0085-2538

VL - 74

SP - 9

EP - 11

JO - Kidney International

JF - Kidney International

IS - 1

ER -

HOming in on arteriovenous fistula survival

Abstract

Access to Document

Other files and links

Fingerprint

Cite this