Keyphrases
2-form
11%
5-fluorouracil (5-FU)
100%
5-flurouracil
100%
Activation Loss
11%
Active Form
11%
Apoptosis
33%
B-cell Lymphoma 2 (Bcl-2)
33%
Caspase-3 Activation
11%
Cell Apoptosis
100%
Close Relationships
11%
Combinational Therapy
11%
Cyclin E
11%
Derivates
11%
DNA Fragmentation
11%
Dominant Negative
44%
Expression Level
11%
G2-M Transition
11%
Ha-ras
100%
Human Bladder Cancer Cells
11%
Induced Apoptosis
22%
Mitochondrial Membrane Potential
11%
NIH3T3
11%
Optimal Dose
11%
Overexpression
100%
P21Cip1
11%
Phosphatidylinositol 3-kinase Pathway
11%
Phosphoinositide 3-kinase (PI3K)
22%
Phosphorylation
11%
Rac1
11%
RAF1
22%
Ras Expression
11%
RAS Oncogene
22%
RhoA
11%
Tumor Cells
11%
Tyrosine
11%
Biochemistry, Genetics and Molecular Biology
Bcl-2
50%
Cancer Cell
16%
Caspase 3
16%
Cell Cycle
16%
Cyclin E
16%
DNA Fragmentation
16%
Fluorouracil
100%
Mitochondrial Membrane Potential
16%
Oncogene H Ras
33%
Optimal Drug Dose
16%
Phosphoinositide 3-Kinase
50%
Programmed Cell Death
100%
Sensitization
33%
Tyrosine
16%
Neuroscience
Bcl-2
50%
Caspase 3
16%
Cell Cycle
16%
Cyclin E
16%
DNA Fragmentation
16%
Fluorouracil
100%
Mitochondrial Membrane Potential
16%
Phosphoinositide 3-Kinase
50%
Programmed Cell Death
100%
Immunology and Microbiology
Bcl-2
50%
Cancer Cell
16%
Caspase
16%
Cell Cycle
16%
Cyclin E
16%
DNA Fragmentation
16%
Mitochondrial Membrane Potential
16%
Oncogene H Ras
33%
Optimal Drug Dose
16%
Programmed Cell Death
100%
Sensitization
33%
Tumor Cell
16%
Tyrosine
16%
Pharmacology, Toxicology and Pharmaceutical Science
Bladder Cancer
33%
Caspase 3
33%
Cyclin E
33%
Fluorouracil
100%
Neoplasm
33%
Phosphatidylinositol 3 Kinase
100%