Keyphrases
3-methylcholanthrene
5%
Activated Microglia
100%
Anti-inflammatory Effect
100%
Aryl Hydrocarbon Receptor
100%
B-site
5%
Bidirectional Effects
5%
Brain Disorders
5%
Carbazole
10%
Cerebral Cortex
5%
Chromatin Immunoprecipitation (ChIP)
5%
Co-treatment
5%
Cytochrome P450 1A1 (CYP1A1)
5%
Environmental Factors
5%
Gene Promoter
10%
Gene-deficient Mice
5%
Immune Response
15%
Induced Expression
5%
Inducible Nitric Oxide Synthase
10%
Inflammatory Effects
10%
Inflammatory Response
5%
Lipopolysaccharide
100%
Lipopolysaccharide-induced
40%
MEK1
5%
Microglia
40%
Mouse Brain
5%
Neuroinflammation
10%
Neurotoxicity
5%
Nitric Oxide Synthase Genes
5%
Receptor Activation
5%
Receptor Binding
5%
Shunt
5%
Signaling Pathway
5%
Tumor Necrosis Factor Genes
5%
Tumor Necrosis Factor-α
5%
Neuroscience
Anti-Inflammatory
100%
Aryl Hydrocarbon Receptor
100%
Carbazole
10%
Cell Signaling
5%
Cerebral Cortex
5%
Cytochrome P450 1A1
5%
Epileptic Absence
5%
Immunoprecipitation
5%
Inducible Nitric Oxide Synthase
10%
Lipopolysaccharide
100%
Microglia
100%
Necrosis
10%
Neurotoxicity
5%
Promoter Region
10%
Receptor Binding
5%
Pharmacology, Toxicology and Pharmaceutical Science
Anti-Inflammatory Drug
10%
Antiinflammatory Activity
100%
Aromatic Hydrocarbon Receptor
100%
Brain Disease
5%
Carbazole Derivative
10%
Chromatin Immunoprecipitation
5%
Cytochrome P450 1A1
5%
Epileptic Absence
5%
Inducible Nitric Oxide Synthase
10%
Inflammation
5%
Lipopolysaccharide
100%
Lipopolysaccharide-Induced Neuroinflammation
5%
Methylcholanthrene
5%
Mouse
10%
Nervous System Inflammation
5%
Neurotoxicity
5%
Tumor Necrosis Factor
10%
Immunology and Microbiology
Antiinflammatory Activity
100%
Chromatin Immunoprecipitation
14%
Immune Response
42%
Inflammation Response
14%
Microglia
100%
Mouse
28%
Promoter Region
28%
Receptor Binding
14%
Signal Transduction
14%
Tumor Necrosis Factor
28%